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J. CLAYTON DAVIE, MD; R. CEBALLOS, MD; S. C. LITTLE, MD

Arch Neurol. 1963;9(3):265-272.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The syndrome of ascending paralysis was described by Landry¹ in 1859, and the first report of what is now termed infectious mononucleosis was made by Pfeiffer² in 1889. In 1916 Guillain, Barré, and Strohl³ reported that facial diplegia and a dispro-portionate increase in the cerebrospinal fluid protein might occur in Landry's paralysis. Since 1927 when Draganesco and Claudian⁴ first referred to this as the "Guillain-Barré" syndrome, this term has come into wide us-age. As Guillain himself⁵ later stated that such cases could occur without the "albuminocytologic dissociation," and as it has been repeatedly reported that otherwise identical cases occur without facial diplegia, the term "Guillain-Barré" syndrome seems to have little special merit. Many other terms have been applied to the same condition, including the name "neuronitis."

In 1931 Johansen⁶ and also Epstein and Dameshek⁷ reported that the nervous system might be involved . . . [Full Text PDF of this Article]

Author Affiliations
BETHESDA, MD; INGATON, ONT, CANADA; BIRMINGHAM

Footnotes
Submitted for publication June 5, 1963; accepted June 8.

National Institute of Neurological Diseases and Blindness, National Institutes of Health (Dr. Davie); Neuropathologist, Hotel Dieu Hospital (Dr. Ceballos); Chief, Division of Neurology, Medical College of Alabama (Dr. Little).

This study was supported in part by US Public Health Service grant 2B 5081, National Institute of Neurological Diseases and Blindness.

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