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W. S. MASLAND, M.D.; L. J. BARROWS, M.D.

Arch Neurol. 1962;7(1):64-73.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The Shwartzman phenomenon, as experimentally produced in animals, is a well-known and characteristic condition. Clinically, there are several syndromes whose course, pathology, and association with bacterial toxins strongly recommend them as human counterparts. This case is presented as another clinical example of the generalized Shwartzman phenomenon, with emphasis on the neurologic symptoms and neuropathology. An attempt to unify the diverse experimental observations on this phenomenon has been made which suggests a relationship between acute hemorrhagic leukoencephalitis and thrombotic thrombocytopenic purpura. The gap between these 2 entities can be bridged by the picture of the Shwartzman phenomenon, thus providing a spectrum.

Report of Case

The patient was a 63-year-old married Negro woman, admitted to the University Hospital June 12, 1958, for vomiting and chest pain of increasing severity. She had enjoyed good health until January, 1958, when she started having bouts of nausea and feelings of distension in the left upper . . . [Full Text PDF of this Article]

Author Affiliations
PHILADELPHIA

Footnotes
Received for publication Dec. 28, 1961.

This investigation was carried out during the tenure of a Postdoctoral Fellowship (No. BF-14,326, Dr. Masland) from the National Institute of Neurological Diseases and Blindness, United States Public Health Service.

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