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Alberto Albanese, MD
Instituto di Neurologia Universitá Cattolica

Carlo Colosimo, MD
I Clinica Neurologica Universitá "La Sapienza" Rome, Italy

Andrew J. Lee, MD
Parkinson's Disease Society Brain Tissue Bank Institute of Neurology London, England

Arch Neurol. 1996;53(3):212-213.

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The issues raised by Caraceni and colleagues are complementary to the clinical findings of our article, which is part of a larger project addressing the clinical diagnosis of MSA and of other related parkinsonian syndromes. In this retrospective study¹ we have reviewed the clinical records of patients who received a neuropathological diagnosis of MSA, idiopathic PD, or progressive supranuclear palsy. Most records were taken at a time when MRI was not widely available. In the cases that were studied with MRI, low-field intensity (0.5 T) was used. Owing to the paucity of MRI scans performed, we could not include MRI as a retrospective diagnostic pointer and instead considered computed tomographic scan.

We are fully aware of the relevance of MRI in MSA and have incorporated it among the items that we have prospectively studied since 1989.² The aim of our project was to identify some prospective . . . [Full Text PDF of this Article]

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