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John P. Blass, MD, PhD; Siegfried Hoyer, MD; Roger Nitsch, MD

Arch Neurol. 1991;48(9):961-972.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The task with which the esteemed committee of our society has charged me, that is, the introduction of the discussion on the definition of generalized progressive paresis by the characterization of the important aspects of the condition, can be divided into three parts: (1) consideration of the anatomic criteria, (2) the causes, and (3) the clinical course.

I shall attempt a brief examination of those of the characteristic features of progressive paresis that allow its differential diagnosis from other similar and related conditions, basing my considerations on all three of these aspects. I hope I may be allowed to observe, however, that the limited scope of this short report will allow me to do no more than give an imprecise indication of the clinical picture and that I hope a more exact representation of the whole or of individual parts of the whole will emerge from the discussion.

The first . . . [Full Text PDF of this Article]

Author Affiliations
From the Altschul Laboratory for Dementia Research, Cornell University Medical College at Burke Rehabilitation Center, White Plains, NY (Dr Blass); and the Institute for Pathological and General Neurochemistry Brain Metabolism Work Group, Ruprecht-Karls-Universitaet Heidelberg (Germany) (Drs Hoyer and Nitsch). Dr Nitsch is now with the Department of Neurology, Massachusetts General Hospital, Boston.

Footnotes
Accepted for publication January 31, 1991.

Presented at the annual meeting of the Society of German Alienists, Dresden, East Germany, September 20,1894.

Reprint requests to the Burke Rehabilitation Center, 785 Mamaroneck Ave, White Plains, NY 10605 (Dr Blass).

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