This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Stephen Stolzberg, MD
Portland Neurological Associates PC 2800 N Vancouver, Suite 202 Portland, OR 97227

Arch Neurol. 1990;47(6):621.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Dobkin et al¹ assert that the reductions in cerebral blood flow in patients with unilateral stroke are due to cerebrovascular disease in the symptomatic hemisphere and diaschisis in the contralateral hemisphere. However, their data show that the median cerebral blood flow in the region of the infarct, in the affected hemisphere, and in the contralateral hemisphere or homologous area to the infarct was the same and that all four of these values were below the 95% confidence limit of their normal controls.

The most economical hypothesis is that stroke develops in patients with a diffuse impairment of cerebral blood flow. The depression in cerebral blood flow in the "normal" hemisphere was measured an average of 21 days after stroke, which hardly fits the reversibility criterion for cerebral diaschisis. Cerebral blood flow prior to stroke has already been reported in patients with basilar insufficiency. These studies all . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?