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FUMIHIKO Okada, MD
Health Administration Center Hokkaido University Sapporo 060, Japan

Yoshiko Shintomi, MD
Shintomi Ophthalmic Clinic Sapporo 064, Japan

Arch Neurol. 1990;47(2):127-128.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Acute pandysautonomia is a syndrome characterized by various symptoms of acute paralysis of the sympathetic and parasympathetic functions, apparently without other clinical or neurological abnormalities.^1,2 Recovery is partial or complete after several months. The site of the lesion of acute pandysautonomia has generally been assumed to be located in the postganglionic fibers of both sympathetic and parasympathetic neurons.^1-3 However, it was suggested that, in the second case of Okada et al³ and in the case of Watanabe et al,⁴ lesions were present in the preganglionic or central nervous system, because of poor vascular responses to subcutaneous or intravenous injection of sympathomimetic amines and no pupillary response to a low concentration of cocaine or epinephrine instillation in the eyes. Electroencephalographic abnormalities were described by Andersen et al,⁵ Harik et al,⁶ and Neville and Sladen.⁷ Hopkins et al⁸ described some temporal . . . [Full Text PDF of this Article]

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