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Judith T. Romano, MD; Jack E. Riggs, MD; John B. Bodensteiner, MD; Ludwig Gutmann, MD
Departments of Neurology and Pediatrics West Virginia University Medical Center Morgantown, WV 26506

Arch Neurol. 1989;46(6):607-608.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—The earliest pathologic changes in moyamoya syndrome are stenotic and/or occlusive changes in the terminal portion of the internal carotid artery.¹ The cause of moyamoya syndrome, particularly the influence of congenital and/or acquired factors, remains undefined. Suzuki² has been the chief proponent of a pathogenetic mechanism that involves the interaction of the autonomic nervous system (local factor) and the immune system (systemic factor). We report wasp sting-associated occlusion of the terminal internal carotid artery, and suggest that this may have similar implications regarding the pathogenesis of moyamoya syndrome.

Report of a Case.

—A previously healthy, developmentally normal 34-month-old white boy was stung repeatedly on the inner upper lip near the midline. The father removed a live yellow jacket from the child's mouth. There were no stings in the posterior aspect of . . . [Full Text PDF of this Article]

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