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Guillermo A. De León, MD
Guillermo DE León Department of Neurology

Warren D. Grover, MD
Department of Pediatrics

Nayere Zaeri, MD
Department of Anatomic Pathology

Philip D. Alburger, MD
Department of Surgery Temple University School of Medicine Philadelphia, PA 19140

Arch Neurol. 1989;46(5):479.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—We wish to reply to the letter to the editor by Jeret et al¹ that appeared in the January issue of the ARCHIVES.

It was clearly stated in our article² that agenesis of the corpus callosum is not a consistent feature of Apert syndrome, and that the callosal malformation is obviously not required to define the disease. We suggested that limbic malformation, with or without agenesis of the corpus callosum, is "probably more common in Apert syndrome than previously thought." The early development and the wide-spread nature of the basic pathologic process were also emphasized in our report. Therefore, it seems that the comments by Jeret et al are superfluous, and that the main purpose of their letter is simply to call attention to the future publication of their two articles on callosal agenesis. We look forward to reading them. . . . [Full Text PDF of this Article]

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