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E. Jeffrey Metter, MD
National Institute on Aging Gerontology Research Center Baltimore, MD 21224

Wayne R. Hanson, PhD; Catherine A. Jackson, MA
Veterans Administration Medical Center Sepulveda, CA 91343

Daniel Kempler, PhD
University of Southern California School of Medicine Los Angeles, CA 90033

Arch Neurol. 1989;46(12):1272-1273.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In Reply.

—The letter by Dr Mark challenges our work on four grounds: (1) the adequacy of the Western Aphasia Battery, (2) the value of resting glucose metabolism in predicting regional dysfunction, (3) whether our conclusions regarding conduction aphasia are warranted, and (4) the failure to use dynamic subtraction studies. We will address these points individually.

The purpose of our two articles^1,2 was to relate glucose metabolic patterns to traditional clinical aphasic syndromes, and not to use the Western Aphasia Battery³ as a means of determining underlying neurolinguistic disturbances. The Western Aphasia Battery was developed to provide a standardized procedure to assist in clinical aphasia classification by providing fixed criteria, thus allowing for a consistent and repeatable diagnosis. Dr Mark is correct in pointing out that the test was not designed to probe the underlying "neurolinguistic" basis of aphasia. Neurolinguistic analyses are particularly important in determining the nature of the . . . [Full Text PDF of this Article]

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