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Julien Bogousslavsky, MD
Department of Neurology CHUV 1011 Lausanne, Switzerland

Arch Neurol. 1988;45(2):143.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Two recent articles have addressed the topographic correlates and the specificity of the syndrome of global aphasia without hemiparesis.^1,2 Tranel et al¹ confirmed the initial article by Van Horn and Hawes,³ because they found that this syndrome was related to two discrete ischemic lesions in the dominant hemisphere (one frontal, one temporoparietal), which were probably of embolic origin. On the other hand, other reports suggested that the syndrome can also be related to a single frontotemporoparietal lesion, which can sometimes be hemorrhagic.^2,4 Unfortunately, this debate is confused by the fact that, although hemiparesis was said to be absent, ten of the 12 described patients had some degree of right-sided weakness or corticospinal

Computed tomographic scan of left hemisphere showing single temporoparietal infarct. dysfunction (increased tendon reflexes, Babinski's sign), or other lateralizing signs (hemianopia). Global aphasia without other lateralizing signs was reported in only . . . [Full Text PDF of this Article]

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