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Peter C. Kohler
University of Vermont College of Medicine Burlington, VT 05405

David Goldblatt, MD
University of Rochester School of Medicine and Dentistry Rochester, NY 14642

Arch Neurol. 1987;44(12):1219.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—We have treated a patient in whom the Guillain-Barré syndrome (GBS) developed 13 days after the onset of febrile diarrhea caused by Campylobacter jejuni. In only seven previously reported cases of GBS has C jejuni been isolated from stool,^1-6 and no such cases (identified either serologically or by culture) have previously been described from countries other than England. Nevertheless, recent reports from England have indicted C jejuni, on serologic evidence, as the "most common single identifiable pathogen precipitating the disease," outranking both cytomegalovirus and Epstein-Barr virus combined.⁷

Report of a Case.

—A 69-year-old man was admitted to a hospital near Watertown, NY, for prostatic surgery and lithotripsy, but those elective procedures were deferred when diarrhea developed, with temperature as high as 39.4°C (103°F). Campylobacter jejuni was cultured from his stool. Erythromycin therapy was instituted, and he was discharged; his diarrhea ceased in about ten days. . . . [Full Text PDF of this Article]

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