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Venkata R. Challa, MD
Department of Pathology Wake Forest University Bowman Gray School of Medicine Winston-Salem, NC 27103

Arch Neurol. 1986;43(6):544.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Reid et al¹ have recently summarized the clinical and pathologic features of pituitary apoplexy, dividing cases of the disorder into those with and without a pituitary tumor. They pointed out that subarachnoid hemorrhage can follow bleeding within a pituitary tumor. I think another important complication of pituitary apoplexy associated with adenomas was omitted. This is the development of intraventricular hemorrhage when hemorrhage arising within a pituitary tumor ruptures through the floor of the third ventricle. In our recent case with this complication,² the outcome was fatal. Our patient had a silent pituitary adenoma; she was undergoing chemotherapy for multiple myeloma. She presented with a sudden onset of coma and died on the same day. At the autopsy she had an endocrine-inactive pituitary adenoma that showed massive necrosis and hemorrhage. The hemorrhage had ruptured through the necrotic capsule and the floor of the third ventricle, filling . . . [Full Text PDF of this Article]

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