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An Indication for Therapeutic Embolization?

John P. H. Wade, MA, MD, MRCP(UK)

Arch Neurol. 1986;43(5):508-509.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Approximately 50% of arteriovenous malformations (AVMs) present with an intracranial hemorrhage,^1-4 and these are predominantly parenchymal.^2,3 The evidence suggests that the risk of hemorrhage in an initially unruptured AVM is 1% to 3% per year, whereas the risk of rebleeding is higher (about 6%) during the first year, although it subsequently drops to just over 2% per year.^5-7 The incidence of occult hemorrhage in resected specimens is high,⁸ and this may be an important factor in the progression of neurological deficit. In addition, there is some evidence that smaller AVMs are more dangerous than giant lesions.^5,7 Although the prognosis for functional recovery is relatively good, with a morbidity and mortality of 10% and 30%, respectively, there is no doubt that deficits can accrue.³

So what has therapeutic embolization got to offer for this most feared complication of AVMs when site and location render surgery . . . [Full Text PDF of this Article]

Author Affiliations
From the National Hospital for Nervous Diseases, London.

Footnotes
Accepted for publication May 28, 1985.

Dr Wade was supported by a fellowship from the Heart and Stroke Foundation of Ontario.

Reprints not available.

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