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  Vol. 42 No. 7, July 1985 TABLE OF CONTENTS
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Cerebral Lateralization

Biological Mechanisms, Associations, and Pathology: III. A Hypothesis and a Program for Research

Norman Geschwind, MD; Albert M. Galaburda, MD

Arch Neurol. 1985;42(7):634-654.

Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Part three of this three-part series commences with other aspects of immunity and infection. Parts one and two appear in previous issues of the ARCHIVES.1,2

OTHER ASPECTS OF IMMUNITY AND INFECTION

In view of the special importance of immune disorders in the anomalous dominance (AD) population, we have devoted a special section to immunity and infection, dealing primarily with some topics not covered earlier.

Localized and Lateralized Immune Effects

In the AD population certain immune disorders occur with especially high frequency, eg, those involving the gastrointestinal tract or the thyroid. There might be several reasons for this, eg, this population might have a high frequency of genes determining special susceptibilities of certain systems. Another possibility is that the elevated frequency of these disorders may be the result of the timing of suppression of the developing thymus gland by hormonal or other effects. During fetal life new antigens are . . . [Full Text PDF of this Article]


Author Affiliations

From the Department of Neurology, Harvard Medical School, Neurological Unit, and Charles A. Dana Research Center, Beth Israel Hospital, Boston, Aphasia Research Center, Department of Neurology, Boston University School of Medicine, and Department of Psychology, Massachusetts Institute of Technology, Cambridge.


Footnotes

Accepted for publication Dec 5, 1984.

{dagger}Deceased.

Reprints not available.



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