• Online Features  This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on Web of Science (1)  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Russell K. Portenoy, MD; Christopher J. Abissi, MD
Department of Neurology

John B. Robbins, MD
Department of Neurosurgery Albert Einstein College of Medicine Bronx, New York 10461

Arch Neurol. 1983;40(9):598.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.

—Impaired venous outflow from the head can cause increased intracranial pressure. Both hydrocephalus (HC)¹ and intracranial hypertension with normal ventricular size² have been described in patients with diminished venous return. We describe a patient with superior vena cava (SVC) obstruction, who had increased intracranial pressure and normal ventricular size.

Report of a Case.

—An 80-year-old man was admitted after an episode of abnormal motor movements. His behavior had recently been characterized by inactivity and periods of staring, as well as several episodes of posturing and unresponsiveness. He had a history of poor vision and heavy alcohol consumption and had been hospitalized once for a possible "stroke."

Initial examination revealed stigmata of cirrhosis and portal hypertension, including hepatosplenomegaly and "spider" angiomata. The patient was alert and oriented and had a mild dementia. His visual acuity was 20/60 bilaterally, with funduscopic findings remarkable only for pale discs. . . . [Full Text PDF of this Article]

CiteULike    Connotea    Delicious    Digg    Facebook    Reddit    Technorati    Twitter     What's this?