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An Experimental Study in Cats

SHAUL FELDMAN, M.D.; ALBERT J. BEHAR, M.D.; DAVID BIRNBAUM, M.D.

Arch Neurol. 1961;4(3):308-317.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Since the first observations made during the last century,²⁸ considerable clinical and experimental evidence has accumulated to support the existence of a relationship between certain intracranial lesions and acute gastrointestional ulcerations and hemorrhages. This evidence was reviewed by Cushing in 1931 in his Balfour Lecture.⁵ He presented a series of patients with intracranial tumors who postoperatively had either gastrointestinal bleeding or perforation. This report stimulated a number of other clinical studies on patients with various pathological conditions of the brain and concomitant gastrointestinal lesions.^11,24,32,37

The neural mechanisms involved in acute gastrointestinal changes have been the subject of investigations since Schiff²⁹ showed in experimental animals that cerebral lesions were associated with gastric ulceration and perforation. This observation was later confirmed by other investigators.^14,17,36 However, many of these lesions were not produced by the stereotaxic method, but by direct surgical procedures which caused damage to other . . . [Full Text PDF of this Article]

Author Affiliations
JERUSALEM, ISRAEL

Department of Nervous Diseases (Laboratory of Experimental Neurology), Department of Pathology and Department of Medicine "A" (Laboratory of Gastroenterological Research), Hadassah University Hospital and the Hebrew University Hadassah Medical School.

Footnotes
Submitted for publication Oct. 20, 1960.

This investigation has been aided by a grant from the Foundations' Fund for Research in Psychiatry.

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