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Remission of Spastic Paraplegia, Dementia, and Optic Neuropathy

Carel Bulens, MD

Arch Neurol. 1981;38(10):669-670.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Neurologic complications of hyperthyroidism are numerous.¹ The patient described here was not known to have thyroid disease and was initially observed to have a combination of spastic paraplegia, dementia, and optic neuropathy. All signs and symptoms improved dramatically within two months of the institution of carbimazole treatment. The optic neuropathy was unusual because there were no clinical or radiological signs of orbital disease.

REPORT OF A CASE

A 72-year-old man was admitted with a six-month history of a slowly progressive gait disturbance, which consisted of weakness of the legs and loss of balance. In addition, he had become confused and uninterested in his surroundings during the last two years. He could not go out alone because he easily lost his way. Both symptoms had greatly worsened during the three weeks before admission. He had become severely apathetic, could not walk, and on several occasions had been incontinent of . . . [Full Text PDF of this Article]

Author Affiliations
From the Department of Neurology, Academic Hospital Rotterdam-Dijkzigt, Rotterdam, the Netherlands.

Footnotes
Accepted for publication Dec 12, 1980.

Reprint requests to Department of Neurology, Academic Hospital Rotterdam-Dijkzigt, Dr Molewaterplein 40, 3015 GD Rotterdam, the Netherlands (Dr Bulens).

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