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J. Chris Sackellares, MD
Department of Neurology University of Michigan University Hospital Ann Arbor, MI 48109

Dennis B. Smith, MD
Department of Neurology Medical College of Georgia Augusta, Ga

Arch Neurol. 1980;37(8):531.

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In Reply.—

We thank Dr Lothman for his comments regarding our report. We agree that the experimental models of Dr Lothman and his co-workers closely approximate our clinical observations. However, we were unable to conclude that the motor activity they described in their "high spinal" preparations was identical to that observed in our patient. The predominant feature of the myoclonus we observed was the absence of any temporal relationship between myoclonic twitches in any given muscle group and twitches observed in other muscle in the ipsilateral or contralateral extremities. Futhermore, the bilateral tonic and clonic movements observed in their animal preparations did not occur in our patient. Because of these differences and lack of diseased material to confirm the degree of brainstem destruction, we could not state unequivocally that the myoclonus originated in the spinal cord. . . . [Full Text PDF of this Article]

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