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S. H. Subramony
Department of Neurology University of Mississippi Medical Center Jackson, MS 39216

A. J. Wilbourn
Department of Neurology Cleveland Clinic Foundation Cleveland, OH 44106

Arch Neurol. 1980;37(10):676-677.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

We read with interest the report on electrophysiologic evaluation in brachial plexus neuropathy by Flaggman and Kelly (ARCHIVES 37:160-164, 1980).

We recently reported a similar study that involved ten patients with idiopathic and three with familial brachial plexopathy.¹ There were no distinguishing features between the groups. Five patients had denervation in predominantly upper trunk distribution, four had diffuse plexus involvement, three had very patchy denervation not easily explained by circumscribed lesions of the brachial plexus, and one had denervation in muscles supplied by the posterior cord. Nerve conduction studies (NCSs) in six patients gave abnormal results, comprising low amplitude of evoked sensory or compound muscle action potentials, together with normal conduction velocities in the elbow to wrist segments. In one of our patients, sensory nerve action potentials were intact when muscles sharing their segmental and peripheral innervation were severely denervated. Some patients showed unusual patterns of . . . [Full Text PDF of this Article]

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