This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

F. Boller, MD
Neurobehavior Unit Cleveland VA Hosp Cleveland, OH 44106

J. M. Segarra, MD
Dept of Neuropathology Boston VA Hosp Boston, MA 02130

Arch Neurol. 1979;36(4):255.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Pogacar et al reported in the ARCHIVES (35:156, 1978) clinical findings in two members of a family with dominant spinopontine atrophy (DSPA), an entity originally described by us in a study of two other members of the same family.¹ On the basis of one postmortem examination, Pogacar et al concluded that DSPA is not clearly distinguishable from olivopontocerebellar atrophy (OPCA). Microscopic examination of the cerebellum and the inferior olivary nuclei of their case undoubtedly showed greater changes than those that occurred in our propositus and in other cases noted in the literature.^2.3 The reason for this difference is unclear; however, it is well known that there can be some variation among members of the same family in the pathological features of spinocerebellar atrophies. The description of Pogacar et al suggests cerebellar and olivary lesions considerably less prominent than those found in classical OPCA, in which . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?