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E. Wayne Massey, MD
Department of Neurology Department of the Navy National Naval Medical Center Bethesda, MD 20014

Arch Neurol. 1979;36(11):727.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor:—

I was interested in the letter by Vallat and Dunoyer in the May 1979 issue of the ARCHIVES (36:323, 1979) about the familial occurrence of carpal tunnel syndrome (CTS). I agree that familial CTS is not rare. Indeed, at Johns Hopkins Hospital (Baltimore), 17% of CTS cases had familial incidence.¹ (Theirs may be a special situation since they are a genetic referral center.)

Another point to be made is that patients with CTS from amyloidosis do not always have amyloid in the flexor retinaculum. Lambird and Harmann¹ found it in seven of ten patients with amyloidosis, and Mahloudji et al² found it in eight of 12 patients with amyloidosis. I have two patients with CTS in whom an ultra-structural examination and Congo red stain of the ligaments did not show evidence of amyloid, but in whom rectal biopsy specimens showed amyloid.

I think that . . . [Full Text PDF of this Article]

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