This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

R. R. Young, MD
Laboratory of Clinical Neurophysiology Massachusetts General Hospital Boston, MA 02114

Arch Neurol. 1977;34(11):722-723.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

To the Editor.—

Tarsy and colleagues (ARCH NEUROL 34:446, 1977) reported the clinical history of a patient with a complicated neurological picture, because one aspect of it included unilateral asterixis. Their patient had been institutionalized before the most recent acute illness because of a long-standing postsubarachnoid hemorrhage hydrocephalic-like disorder with decreased memory, signs of "frontal lobe release," and an abnormal EEG. After developing asterixis, he was said to have "no significant sensory deficits," though they reported he did have reduced two-point discrimination in the hand affected with asterixis. The presence of this deficit weakens the authors' argument that the unilateral asterixis described by Leavitt and Tyler¹ in association with unilateral disturbances in proprioceptive input is in some way different from that seen in the patient of Tarsy and his colleagues. The latter also dismiss as unlikely the suggestion that the hemiparetic weakness their patient sustained was directly related . . . [Full Text PDF of this Article]

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?