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Glucose and Baroreceptor FunctionEffects of Oral Administration of Glucose on Baroreceptor Function in Cerebrovascular Disease and in Other Disorders With Baroreceptor Reflex Block
Otto Appenzeller, MD, PhD;
J. E. Goss, MD
Arch Neurol. 1970;23(2):137-146.
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THE OCCURRENCE of "strokes" in association with meals is well recognized, but this relationship is not fully understood. One postulated mechanism is that patients lose vasomotor control after a large meal so that a change in posture after the feast leads to significant falls in blood pressure, jeopardizing blood flow in an already diseased part of the vasculature of the brain, with consequent infarction. This hypothesis has some support. It has been shown, for example, that elderly subjects have defective vasomotor control and compensate poorly for sudden changes in cardiac output.1,2 It is possible, therefore, that in such subjects further impairment of vasomotor function related to the meal may significantly decrease cerebral blood flow in a compromised part of the vasculature.
Some patients with diabetes, with or without clinically recognizable peripheral neuropathy, have impaired baroreceptor reflexes.3 In such patients and in others without diabetes but with impaired
. . . [Full Text PDF of this Article]
Author Affiliations
Albuquerque, NM
From the departments of neurology (Dr. Appenzeller), and medicine (Drs. Appenzeller and Goss), University of New Mexico Medical School, Albuquerque, NM.
Footnotes
Submitted for publication Dec 6, 1969; accepted Jan 13, 1970.
Read before the American Neurological Association, Los Angeles, June 9-11, 1969.
Reprint requests to Department of Neurology, University of New Mexico Medical School, Albuquerque, NM 87106 (Dr. Appenzeller).
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