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Robert H. Wilkins, MD; Irwin A. Brody, MD

Arch Neurol. 1970;22(3):279-280.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IN THE Edwin Smith Surgical Papyrus, dating from the 17th century BC, an association was noted between temporal skull trauma and loss of speech.¹ Nevertheless, before the work of Broca, the brain was generally thought to act as a whole, with no anatomical localization of the different cerebral functions.^2,3 In 1861, Paul Broca presented two cases of dysphasia associated with lesions in the posterior portions of the second and third left frontal convolutions.^4,5 Although Broca underestimated the extent of the lesions in his first patient, his provocative presentations provided an early anatomical localization for the expressive aspects of speech.³

In 1874, the German neuropsychiatrist, Carl Wernicke (1848 to 1905),^6-9 published a short monograph in which he used simple anatomical diagrams to present a more comprehensive view of speech mechanisms.¹⁰ Wernicke described five clinical syndromes that would be expected from lesions of (1) the . . . [Full Text PDF of this Article]

Author Affiliations
Durham, NC

From the Divisions of Neurosurgery and Neurology, Duke University Medical Center, and the Durham Veterans Administration Hospital, Durham, NC.

Footnotes
Submitted for publication Oct 28, 1969; accepted Nov 1.

Reprint requests to Division of Neurosurgery, Duke University Medical Center, Durham, NC 27706 (Dr. Wilkins).

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