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With Type II Muscle Fiber Hypotrophy

George H. Cardinet III, DVM, PhD; Marion R. Fedde, MS, PhD; John W. Bardens

Arch Neurol. 1969;21(6):620-630.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

HIP DYSPLASIA in dogs is a hereditary disease, common to many large breeds; a notable example is the German shepherd breed in which as many as 50% of the dogs are affected.^1-4 Dogs which will develop the disease cannot have the condition diagnosed at birth. In most instances, radiographic diagnosis is not possible until after 8 weeks of age and in mild cases not until 6 to 12 months.^3,4

Though the etiology of hip dysplasia in the dog is unknown, the wide variety of acetabular and femoral changes observed appear to be the consequence of joint laxity.³ Hence, studies of factors affecting joint laxity offer promise of determining the etiology of the disease. Recently, a technique was developed to adjudge laxity of the hip joint in puppies.⁵ In the development of this technique, it was observed that as early as 4 weeks of age, in . . . [Full Text PDF of this Article]

Author Affiliations
From the Neuromuscular Research Laboratory, Department of Physiological Sciences, College of Veterinary Medicine, Kansas State University, Manhattan, Kan.

Footnotes
Submitted for publication June 13, 1969; accepted July 1.

Reprint requests to Neuromuscular Research Laboratory, Department of Physiological Sciences, Laboratory, College of Veterinary Medicine, Kansas State University, Manhattan, Kan 66502 (Dr. Cardinet).

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