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Clinical and Electromyographic Considerations

R. D. TEASDALL, M.D.; M. L. SEARS, M.D.

AMA Arch Neurol. 1960;2(3):281-292.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Although primary involvement of the ocular muscles was recognized as a distinct entity many years ago by Gowers,¹ other observers have followed Moebius'² claim that the resulting weakness was due to nuclear degeneration. In 1951, however, Kiloh and Niven³ extensively reviewed the pertinent literature, presented five cases, and concluded that these entities were similar in their pathology, namely, primary disease of the ocular muscles. These observations have since been confirmed by biopsy of involved muscle^4,5 and from autopsy material.^6,7 The term ocular myopathy is now, therefore, in general usage for such cases.

In this study, the clinical, and in particular the electromyographic, findings are presented in six patients with the diagnosis of ocular myopathy.

I. Clinical Findings

Report of Cases

Case 1.

—Ten years ago, this 54-year-old patient first noticed drooping of the right lid and double vision. Examination revealed complete right ptosis, without compensatory . . . [Full Text PDF of this Article]

Author Affiliations
Baltimore

From the Division of Neurological Medicine and the Wilmer Institute, The Johns Hopkins University School of Medicine and Hospital.

Footnotes
Received for publication Oct. 8, 1959.

Supported by a grant (B-367) from the National Institute of Neurological Diseases and Blindness, National Institutes of Health, U.S. Public Health Service.

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