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Neuropathy in MyxedemaA Study of Sensory Nerve Conduction in the Upper Extremities
R. W. Fincham, MD;
C. A. Cape, MD
Arch Neurol. 1968;19(5):464-466.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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THE carpal tunnel syndrome, a compression neuropathy, has been accepted as a neurological complication of myxedema.1,2 An intrinsic neuropathy secondary to myxedema has been suggested but not as yet clearly proven.3 In an effort to elucidate this latter condition a study of median and ulnar sensory nerve potentials has been done in myxedematous patients.
Methods
Sixteen patients with an unequivocal diagnosis of myxedema established by clinical and laboratory criteria were included in this survey. Their ages ranged from 30 to 84 years (average age 56). Median and ulnar evoked sensory nerve potentials were sought in all of these patients in a manner similar to that outlined by Dawson and Scott.4 Stimulating electrodes were placed about the second and third digits for median nerve determinations and about the fourth and fifth digits for ulnar nerve determinations. The stimulating cathode was located at the metacarpal phalangeal joints while the
. . . [Full Text PDF of this Article]
Author Affiliations
Iowa City
From the Department of Neurology and the Neurosensory Center, College of Medicine, University of Iowa, Iowa City. Dr. Cape is currently at the Department of Neurology, the University of Tennessee.
Footnotes
Submitted for publication June 11, 1968; accepted July 29.
Reprint requests to Department of Neurology, University of Iowa College of Medicine, Iowa City 52240 (Dr. Fincham).
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