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Report of Cases

Joseph B. Martin, MD; Randall H. Travis, MD; Stanley van den Noort, MD

Arch Neurol. 1968;19(2):163-173.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

ORTHOSTATIC hypotension may be primary or secondary. The clinical syndrome of primary idiopathic orthostatic hypotension is characterized by inability to sustain standing blood pressure without the development of pallor or of tachycardia.¹ Other findings include anhidrosis, impotence, urinary urgency or retention, alternating constipation and diarrhea, and a variety of disturbances directly pointing to disease of the central nervous system.² Evidences of central nervous system disease include dysarthric or monotonous speech, diplopia, pupillary irregularities, vertigo, rigidity, tremor, cerebellar ataxia, and incontinence.²

Orthostatic hypotension, as a secondary phenomenon, occurs in a wide variety of disease states and after bed rest. Interest in orthostatic hypotension has been stimulated by its development after space flights.³ Sustained orthostatic hypotension has been described in neuropathies (diabetic,⁴ porphyric,⁵ nutritional), in myasthenia gravis,⁶ in paraplegia of varied causes, in Wernicke's encephalopathy,^7,8 in combined systems disease,⁹ in tumors of . . . [Full Text PDF of this Article]

Author Affiliations
Cleveland

From Case Western Reserve University School of Medicine, Department of Medicine, Division of Neurology (Drs. Martin, Travis, and van den Noort) and the Department of Physiology (Dr. Travis) and University Hospitals (Drs. van den Noort and Travis), Cleveland. Dr. Martin is now at the University of Rochester Medical School, Endocrine Unit, Rochester, NY.

Footnotes
Submitted for publication Dec 18, 1967; accepted Feb 17, 1968.

Reprint requests to Division of Neurology, University Hospitals, Cleveland 44106 (Dr. van den Noort).

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