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A Syndrome of Intracranial Hypertension

Maurice W. Van Allen, MD

Arch Neurol. 1967;17(1):81-88.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Paralysis of the abducens nerve may be a consequence of intracranial hypertension of whatever cause. The vulnerability of this nerve has been ascribed to stretching and bony impingement with downward displacement of the brain stem,^1,2 to notching by branches of the basilar artery,³ and, in a general way, to its long unprotected course. The loss of ocular abduction, which is equated with paralysis of the nerve, while subject to some variation, is usually persistent over a period of days or weeks.

The opportunity to observe transient and recurring paralysis of ocular abduction in two patients suffering from intracranial hypertension has prompted this description of a phenomenon which has received limited attention in the past.^3-7 The fluctuating level of function in this condition might be explained by proposing short-lived changes in the dynamics of abducens paralysis noted above. However, the associated symptoms in one of the patients . . . [Full Text PDF of this Article]

Author Affiliations
Iowa City

From the Department of Neurology, College of Medicine, University of Iowa, and the Neurosensory Center, Iowa City.

Footnotes
Submitted for publication Nov 7, 1966; accepted Feb 9, 1967.

Read by title before the 90th meeting of the American Neurological Association, Atlantic City, NJ, June 14-16, 1965.

Reprint requests to Department of Neurology, University Hospital, Iowa City 52240 (Dr. Van Allen).

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