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WILLIAM M. LANDAU, MD; MARGARET H. CLARE, MA

Arch Neurol. 1966;15(3):252-263.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

THE VARIETIES of exaggerated flexion movements in the hand resulting from forebrain lesions have received much detailed study.^1-7 Most authors have pointed out that related flexion in the foot may be present in some patients, but less investigation has been focused upon the lower extremity, and one may infer that this is partially because the phenomenon is less conspicuous there.

Wilson and Walshe¹ described a behavior of prolonged innervation of the finger flexors which the patient was unable to relax. Adie and Critchley³ emphasized the reflex nature of pathological grasping but indicated that it may be triggered by voluntary effort. Later studies by Walshe^4,5 concluded that there are two phenomena, one an exaggerated volitional contraction and the other a reflex response to proprioceptive stimulation of the fingers. A similar reflex was also described in the foot.

The most detailed study is that of Seyffarth and . . . [Full Text PDF of this Article]

Author Affiliations
ST. LOUIS

From the Department of Neurology and the Beaumont-May Institute of Neurology, Washington University School of Medicine, St. Louis.

Footnotes
Submitted for publication May 5, 1966; accepted May 19.

Reprint requests to Washington University School of Medicine, 660 S Euclid Ave, St. Louis 63110 (Dr. Landau).

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