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BENNETT M. STEIN, MD; MALCOLM B. CARPENTER, MD

Arch Neurol. 1965;13(6):567-583.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

ONE UNRESOLVED question concerning various forms of dyskinesia is the role that sensory input plays in the underlying basic neural mechanism. Relatively little data are available concerning the effects of dorsal rhizotomies upon human dyskinesia mainly because this procedure would have to be extensive and would result in widespread sensory loss. In the human, dorsal rhizotomy has been performed primarily for relief of pain, visceral crises, and spasticity or rigidity.^{10,13,15,16,18,33} There are, however, a few instances in which dorsal rhizotomies have been performed upon patients with dyskinesia. According to Frazier,¹⁵ surgical section of successive cervical dorsal roots in a patient with infantile diplegia caused spasticity to be diminished and all athetoid movements to disappear. A similar result was reported by Clark and Taylor¹⁰ but all spasticity was abolished in this case. Foerster,¹³ whose extensive experience with dorsal rhizotomy is well known, summarized his views on resection . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Department of Anatomy, College of Physicians and Surgeons, Columbia University, New York.

Footnotes
Submitted for publication July 27, 1965; accepted Sept 13.

Reprint requests to Department of Anatomy, College of Physicians and Surgeons, Columbia University, 630 W 168th St, New York, NY 10032 (Dr. Carpenter).

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