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Hypertensive Encephalopathy
DEWEY K. ZIEGLER, MD;
ANGELO ZOSA, MD;
TURGUT ZILELI, MD
Arch Neurol. 1965;12(5):472-478.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings. |
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THE CLINICAL entity of seizures and focal neurological signs and symptoms complicating advanced hypertension was formulated as "hypertensive encephalopathy" by Fishberg 40 years ago, and ascribed to a specific mechanism of arterial spasm and cerebral edema. It has been our impression that the cerebral manifestations of many of the cases so diagnosed might well be explained by more familiar and well defined disturbances of brain physiology— such as hemorrhage, infarction, vascular insufficiency, uremia, or electrolyte imbalance. It was therefore decided to review an unselected series of cases diagnosed as hypertensive encephalopathy and to critically evaluate the findings: (1) to determine the accuracy with which Fishberg's diagnostic criteria were met; and (2) to determine if the actual mechanism of brain dysfunction in retrospect was possibly other than angiospasm.
Material and Methods
All cases with a final diagnosis of hypertensive encephalopathy discharged from the Kansas University Medical Center over a ten year
. . . [Full Text PDF of this Article]
Author Affiliations
LOS ANGELES; ANKARA, TURKEY
From the Division of Neurology, Department of Medicine, Kansas University Medical Center.
Footnotes
Submitted for publication Nov 23, 1964.
Dr. Zileli's Present address, Hacettepe Tip Enstitusu, Fakultesi, Ankara, Turkey (Dr. Zileli).
A portion of this work was published in the Transactions of the American Neurological Association, 1962.
Reprint requests to Rainbow Blvd at 39th St, Kansas City, Kan 66103 (Dr. Ziegler).
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