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  Vol. 11 No. 3, September 1964 TABLE OF CONTENTS
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Ocular Electromyography in Brain Stem Dysfunction

Clinical and Electrophysiological Analysis of a Case With Multiple Eye Movement Disorders

RICHARD N. REUBEN, MD; CALEB GONZALEZ, MD

Arch Neurol. 1964;11(3):265-272.

Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The purpose of this paper is to show how electromyography (EMG) of the eye muscles serves to elucidate physiologic phenomena occurring in the presence of brain stem dysfunction.

One of the basic tenets of ocular muscle physiology is that excitation of one set of muscles (agonists) is associated with the inhibition of the opposing set (antagonists), exemplified both ipsilaterally and contralaterally. Electromyography affords an opportunity to demonstrate these basic phenomena not only in the course of normal eye movements but also in conjugate and disconjugate gaze disorders and even, as in this case, in apparent (clinical) paralysis of eye movement. While many descriptions of ocular motor disorders are available, most reports discuss the defects in eye movement without specifically alluding to excitation or, in particular, to inhibition. This report will emphasize these phenomena and the correlation of clinical and electromyographic findings.

From the electrophysiologic point of view, one of the . . . [Full Text PDF of this Article]


Author Affiliations

NEW YORK

From the departments of neurology, pediatrics, and ophthalmology, New York University School of Medicine.


Footnotes

Submitted for publication April 3, 1964; accepted April 4.

Special Research Fellow, BT-577, currently Director of Pediatric Neurology, New York University School of Medicine (Dr. Reuben); Special Research Fellow, NB-983-02, from the National Institute of Neurological Diseases and Blindness, United States Public Health Service, present address: Department of Ophthalmology, University of Puerto Rico School of Medicine, San Juan, Puerto Rico (Dr. Gonzalez).

This study was supported in part by research grants from the National Institute of Neurological Diseases and Blindness, NB-00911, United States Public Health Service, and the National Society for the Prevention of Blindness, Inc.



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