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A Study of 13 Cases

FLAVIU C. A. ROMANUL, MD; ARTUR ABRAMOWICZ, MD

Arch Neurol. 1964;11(1):40-65.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Introduction

Selective infarction of the portions of brain situated in border zones between the territories of supply of the major cerebral arteries has been described in the past on several occasions and has been attributed to a variety of disease processes. Pathological changes in small pial vessels over the areas of infarction were encountered in some of these cases, and these were also interpreted differently by various authors. We wish to present evidence of what we believe to be a common mechanism responsible for the pathological changes in all such cases.

In 1934 Pentschew¹ studied nine brains with granular atrophy of the cerebral cortex. The brain damage in these cases had been attributed to several causes including atherosclerosis. In all the brains the granular atrophy affected the same parts of the cerebral cortex: the middle frontal gyri, the upper portions of the pre- and postcentral gyri, the superior parietal . . . [Full Text PDF of this Article]

Author Affiliations
BOSTON

From the Neurological Unit, Boston City Hospital and the Department of Neurology, Harvard Medical School.

Footnotes
Submitted for publication Nov 28, 1963; accepted Feb 20, 1964.

Supported in part by a research grant (NB 03477-03) from the National Institute of Neurological Diseases and Blindness. The electron microscopic study was supported by a grant (AM-04165) from the National Institutes of Arthritis and Metabolic Diseases, National Institutes of Health, Bethesda, Md.

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