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MURRAY A. FALCONER, MCh, FRCS; EUSTACE A. SERAFETINIDES, MD, DPM; J. A. NICHOLAS CORSELLIS, MRCS, LRCP

Arch Neurol. 1964;10(3):233-248.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Increasing attention is being paid to the relationship of cerebral lesions to the epilepsies. Gastaut and Fischer-Williams²⁷ consider that cases of so-called functional or idiopathic epilepsy (which represent a fault in cerebral function rather than in structure) constitute only about 5% of cases and that most cases of epilepsy have underlying cerebral lesions. These lesions, however, may develop on a "soil" already predisposed to convulsions, which enables a mildly irritative lesion to become markedly epileptogenic. Gastaut^24-26,28 has also reviewed our present knowledge of the causes of temporal lobe epilepsy, which is a common form of focal or "partial" epilepsy.^12,24,35,51 However, this knowledge is still incomplete, and as Yakovlev⁶⁶ has pointed out, various lesions may be responsible. In this paper, therefore, we record the pathological findings in 100 consecutive patients with epilepsy submitted to a unilateral temporal lobectomy, and discuss their origin and their significance.

Case . . . [Full Text PDF of this Article]

Author Affiliations
LONDON

From the Guy's-Maudsley Neurosurgical Unit and the Department of Neuropathology, Institute of Psychiatry, Maudsley Hospital.

Footnotes
Submitted for publication Sept 23, 1963; accepted Dec 6.

Financial support has been forthcoming from the Medical Research Council and from the Research funds of Guy's Hospital and of the Bethlem Royal and Maudsley Hospitals.

A personal grant from Guy's Hospital Medical School (Dr. Serafetinides).

Read in part at the Second European Congress of Neurosurgery, Rome, Italy, April, 1963.

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