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JEROME DERSH, M.D.; NATHAN S. SCHLEZINGER, M.D.

AMA Arch Neurol. 1959;1(6):695-699.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Occasionally the ophthalmologist or the neurologist is confronted with a patient with bilateral papilledema associated with increased intracranial pressure for which no cause can be found, even after extensive neurologic examination and neurosurgical intervention. The signs and symptoms indicative of increased intracranial pressure simulate those of a cerebral tumor, and therefore the term "pseudotumor cerebri" has been applied to this condition. This syndrome previously has been referred to in the literature as otitic hydrocephalus, serous meningitis, intracranial hypertension, generalized cisternal archnoiditis, and toxic hydrocephalus.^1,3,10

In many patients with pseudotumor cerebri no abnormality in visual fields is found. Of the field defects reported, the commonest is the enlargement of the blind spots.² Foley,⁹ in his series of cases of pseudotumor cerebri, observed patients with homonomous quadrant defects, central scotomas, and constricted fields. Dunn and Baker⁷ reported a case of pseudotumor cerebri that showed an inferior nasal quadrantanopsia . . . [Full Text PDF of this Article]

Author Affiliations
Philadelphia

From the Departments of Neurology, Wills Eye Hospital and Jefferson Medical College.

Footnotes
Accepted for publication July 1, 1959.

Presented at the 86th Annual Meeting of the American Neurological Association in Atlantic City, on June 16, 1959.

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