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Report of a Case Resistant to Dextrose and Insulin Provocation

RAYMOND F. CHEN, M.D.

AMA Arch Neurol. 1959;1(5):475-484.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Familial periodic paralysis is a disease of unknown etiology characterized by recurrent acute attacks of weakness associated with a fall in serum potassium levels. The syndrome of acute and/or repeated paresis, however, can be brought on by a variety of factors, which must be distinguished from those in familial periodic paralysis. Hammes¹ considers that the commonest causes of acute paresis are poliomyelitis, hysteria, the Guillain-Barré syndrome, post-diphtheritic neuritis, tic paralysis, porphyria, and necrotizing myelitis. In addition, he reports three cases of familial periodic paralysis in which the disease was misdiagnosed as poliomyelitis, malingering, and myasthenia gravis, respectively.

Muscular weakness may result from both hypokalemia and hyperkalemia. Milne, Stanbury, and Thomson² observed a patient with the Fanconi syndrome who lost potassium excessively and whose condition had actually been diagnosed as familial periodic paralysis until the true picture was revealed. Attacks of flaccid paralysis were seen in the patients . . . [Full Text PDF of this Article]

Author Affiliations
New York

From the Second (Cornell University) Neurologic Service, Bellevue Hospital, and Cornell University Medical College.

Footnotes
Received for publication June 15, 1959.

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