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Babak B. Navi, MD; Kathy Kawaguchi, MD; Ingrid Hriljac, MD; Ehud Lavi, MD; Lisa M. DeAngelis, MD; Dara G. Jamieson, MD

Arch Neurol. 2009;66(9):1174-1175.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A 49-year-old woman presented with 2 weeks of cough and fevers and 1 week of right-sided weakness and difficulty speaking. Examination was notable for tachycardia, distant heart sounds, jugular venous distention, pulsus paradoxicus, and Janeway lesions. Neurologic examination revealed global aphasia, right homonymous hemianopsia, and a right hemiparesis. Laboratory examination was remarkable for a troponin level of 0.45 ng/mL (to convert to micrograms per liter, multiply by 1) and an erythrocyte sedimentation rate of 83 mm/h. Head computed tomography demonstrated an acute left middle cerebral artery territory infarction (Figure, A). Transthoracic echocardiogram showed a pericardial effusion, cardiac tamponade, and 3 large mobile echodensities in the left ventricle (Figure, B). Chest computed tomography demonstrated a large mediastinal mass encasing the great vessels and bronchi, innumerable pulmonary . . . [Full Text of this Article]

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RELATED LETTERS

Janeway Lesions, Osler Nodes, or Neither?
Weekitt Kittisupamongkol
Arch Neurol. 2010;67(3):373.
EXTRACT | FULL TEXT  

Janeway Lesions, Osler Nodes, or Neither?—Reply
Babak B. Navi and Lisa M. DeAngelis
Arch Neurol. 2010;67(3):373.
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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Janeway Lesions, Osler Nodes, or Neither?
Kittisupamongkol
Arch Neurol 2010;67:373-373.
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Janeway Lesions, Osler Nodes, or Neither?--Reply
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Arch Neurol 2010;67:373-373.
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