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Panagiotis Papanagiotou, MD; Tilman Rohrer, MD; Iris Q. Grunwald, MD; Maria Politi, MD; Ludwig Gortner, MD; Wolfgang Reith, MD

Arch Neurol. 2009;66(7):906-907.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A 4-month-old infant was referred to the pediatrics neurology clinic with macrocephaly. Her neurological status was normal. Sonography, magnetic resonance imaging (MRI) and angiography revealed a large vein of Galen aneurysmal malformation (VGAM) (Figure 1). The patient was treated endovascularly, using ethylene-vinyl alcohol copolymer (Onyx) as an embolic agent. Occlusion of the feeding arteries was complete, and there was no remaining flow into the VGAM (Figure 2). Follow-up MRI after 1 year showed that hydrocephalus persisted, whereas the VGAM had shrunk (Figure 2). The patient had development delay and seizures.

Figure appears in full text version. Figure 1. A, Doppler sonography depicts 2 arterial feeders (arrows) and circular blood flow within the cyst, which demonstrates the vascular nature of the lesion. T2-weighted cranial magnetic resonance imaging in sagittal and transversal views . . . [Full Text of this Article]

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