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Katharina Hüfner, MD; Markus Holtmannspötter, MD; Hartmut Bürkle, MD; Ulrich C. Schaller, MD; Anne D. Ebert, PhD; Heiko Trentzsch, MD; Hans-Walter Pfister, MD; Christoph B. Lücking, MD

Arch Neurol. 2008;65(8):1124-1125.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A 16-year-old girl hit her left leg against a tree while riding horseback. At admission to a community hospital, a tibial wedge fracture was diagnosed and treated conservatively (Figure, A). Twenty hours after the trauma, the patient became nauseous and vomited. Three hours later, automotor seizures occurred, and she became comatose. Endotracheal intubation was performed to protect the airway (Glasgow-Coma Scale 7/15). A diagnostic workup including drug screening, lumbar puncture, standard laboratory testing, 2 cerebral computed tomographic scans, and cerebral computed tomographic angiography revealed no pathologic findings.

Figure appears in full text version. Figure. Radiograph of the tibial fracture (A) and typical manifestations of fat embolism syndrome (B-E, G, and H). Note cotton-wool spots on the retina (B) and rash on the trunk (C). Diffusion-weighted magnetic resonance . . . [Full Text of this Article]

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