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  Vol. 65 No. 7, July 2008 TABLE OF CONTENTS
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Primary Brain Amyloidoma

Long-term Follow-up

Dimitri Renard, MD; Chantal Campello, MD; Valerie Rigau, MD; Nicolas de Champfleur, MD; Pierre Labauge, MD, PhD

Arch Neurol. 2008;65(7):979-980.

Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A 39-year-old woman presented with generalized seizures. Clinical examination findings were normal. Brain computed tomography showed 2 hyperdense subcortical lesions in the right temporal and frontal lobes. Enhancement was seen in the temporal lesion after contrast injection. Magnetic resonance imaging patterns were as follows: the temporal lesion was slightly hypointense on the T1-weighted sequence, clearly enhanced by gadolinium administration (Figure 1A), and hypointense surrounded by a hyperintense signal on the T2-weighted sequence; the frontal lesion was slightly hypointense on the T1-weighted sequence, enhanced after gadolinium injection (Figure 1A), and mixed isointense/hyperintense on the T2-weighted sequence. Findings from the cerebral spinal fluid examination, cerebral angiography, mammography, and thoracic, abdominal, and pelvic computed tomography were normal. A multifocal brain tumor was suspected. Carbamazepine therapy was started. Four years later, the signal . . . [Full Text of this Article]

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THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES

Brain Amyloidoma With Cerebral Hemorrhage
Labro et al.
JAOA: Journal of the American Osteopathic Association 2009;109:372-375.
ABSTRACT | FULL TEXT  





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