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Arch Neurol. 2006;63:1026-1027.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

When Wernicke encephalopathy (WE) is associated with gastric surgery, it usually develops weeks or months after the procedure. Its development more than 20 years after surgery is extremely rare and, to our knowledge, all the reported cases pertain to Japanese patients.^1-2 Moreover, magnetic resonance images were remarkable in a single patient.² We describe the first white patient who developed WE 28 years after gastric surgery and had reversible lesions shown on magnetic resonance images in all the typical locations where the pathologic abnormalities of WE are known to occur.

Ten days prior to admission, a 53-year-old man gradually developed unsteadiness of gait, a worsening feeling of dizziness, and 2 episodes of vomiting. In 1967, he had undergone partial gastrectomy with gastrojejunostomy because of a peptic ulcer and had been receiving monthly intramuscular injections of vitamin B12. Three years ago, he had axonal sensorimotor polyneuropathy that had gradually improved following multivitamin . . . [Full Text of this Article]

AUTHOR INFORMATION

Theodoros Karapanayiotides, PhD; Ioanna Zaloni, MD; Thomas Thomaides, PhD

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