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Bilateral Paramedian Thalamic Infarcts
Arch Neurol. 2006;63:1652.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. |
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A 60-year-old man with hypertension and dyslipidemia had a sudden onset of somnolence and gait ataxia. The neurological examination revealed skew deviation, bilateral dysmetria, and bilateral extensor plantar responses. Fluctuation in consciousness was noted for several weeks, but the patient gradually recovered. The neuroimaging studies demonstrated symmetric bilateral hyperintense thalamic lesions on T2-weighted images consistent with ischemic events (Figure 1). Basilar arteries and posterior cerebral arteries (PCAs) were fully patent on magnetic resonance angiography (not shown).
Figure appears in full text version.
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Figure 1. T2-weighted image showing the bilateral symmetric thalamic hyperintensities (arrows) of the first patient.
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A 63-year-old man with thalassemia and no risk factors for stroke was admitted for somnolence, confusion, disorientation, and inappropriate social behavior. The neurological examination demonstrated anterograde memory impairment, left facial weakness, and a minimally affected left pyramidal tract. The T2- (Figure 2A) and diffusion-weighted images (Figure 2B) showed bilateral paramedian thalamic infarcts. Digital . . . [Full Text of this Article] COMMENT
AUTHOR INFORMATION
Sotirios Giannopoulos, MD;
Vasiliki Kostadima, MD;
Aikaterini Selvi, MD;
Panagiotis Nicolopoulos, MD;
Athanassios P. Kyritsis, MD
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