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  Vol. 61 No. 12, December 2004 TABLE OF CONTENTS
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  History of Neurology: Seminal Citations
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 •History of Medicine
 •Viral Infections
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 •Herpes
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The Neurology of Varicella-Zoster Virus

A Historical Perspective

Raul G. Nogueira, MD; Bryan Traynor, MD, MRCPI

Arch Neurol. 2004;61:1974-1977.

Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

INTRODUCTION

Varicella-zoster virus (VZV) causes 2 distinctive syndromes: varicella (chickenpox), the primary infection, which is usually a benign epidemical illness from childhood that manifests as a generalized pruritic rash, and herpes zoster (shingles), which results from the reactivation of latent VZV within the sensory ganglia and typically manifests as a painful dermatomal eruption (Figure 1).


 
Figure appears in full text version.
Figure 1. A color lithograph entitled "Herpes Zoster faciei et capitillii" published in Atlas der Hautkrankheiten (Hebra’s Atlas of Skin Diseases) in 1866 by von Carl Heitzmann. (Source: Diepgen TL, Yihune G, et al. Dermatology Online Atlas. http://www.dermis.net/doia/.)


The VZV itself is estimated to have emerged some 70 million years ago.1 The disease known as herpes zoster has been recognized since ancient times and was initially named by Hippocrates (herpes= to creep). Zoster is similarly derived from Greek (girdle), whereas shingles comes from Latin . . . [Full Text of this Article]

ETIOLOGY: THE RELATIONSHIP BETWEEN VARICELLA AND ZOSTER

PATHOGENESIS: THE SENSORY GANGLIA AND VIRAL LATENCY

CYTOPATHOLOGICAL DIAGNOSIS

POSTHERPETIC NEURALGIA

TREATMENT

PROPHYLAXIS

AUTHOR INFORMATION

Author Affiliations: Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Boston.







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