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Landau-Kleffner Syndrome
Joshua Rotenberg, MD;
Phillip L. Pearl, MD
Arch Neurol. 2003;60:1019-1021.
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INTRODUCTION
. . . it may be suggested that persistent convulsive discharge in brain tissue largely concerned with linguistic communication results in the functional ablation of these areas for normal linguistic behavior.—William M. Landau, MD, and Frank R. Kleffner, PhD1
From the personal perspective of attaining three score and seven and the condition of Status Eponymicus, I can find no basis for ego gratification.—William M. Landau, MD2
The description of Landau-Kleffner syndrome (LKS) has profoundly influenced not only clinical practice but also cognitive neuroscience. At the time of their interdisciplinary collaboration, Landau and Kleffner (Figure 1 and Figure 2) could not have known the significance of their description of this rare disorder of communication. Landau has lamented and satirized the achievement of "status eponymicus,"2 given the enduring lack of rigorous data associated with the comingling of acquired aphasia and epilepsy. The central . . . [Full Text of this Article]
FIRST DESCRIPTION
PATHOGENESIS
AN LKS SPECTRUM: OVERLAP SYNDROMES
TREATMENT
OUTCOME
From the Department of Child and Adolescent Neurology, Walter Reed Army Medical Center, Washington, DC, and Uniformed Services University of the Health Sciences, Bethesda, Md (Dr Rotenberg); and Department of Neurology, Children's National Medical Center, George Washington University School of Medicine, Washington, DC (Dr Pearl).
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES
Landau-Kleffner syndrome and temporal cortical volume reduction: Cause or effect?
Bourgeois and Landau
Neurology 2004;63:1152-1153.
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