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Arch Neurol. 2001;58:1345-1347.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

A PATIENT who may or may not have had exposure to Borrelia burgdorferi–infected ticks presents with chronic malaise, a perception of impaired cognition and memory, and depression. These symptoms may or may not have been preceded by classic Lyme disease manifestations. Antibiotics may or may not have been given. Is this disorder chronic central nervous system (CNS) Lyme disease? This diagnosis, often applied in such circumstances, explicitly or implicitly poses 4 questions: Does the patient have Lyme disease? Does the patient have an infection of the CNS (neuroborreliosis)? If the illness is due to Lyme disease, why is there such interindividual variability in disease manifestations? Finally, and most important, if not Lyme disease, what is this disease that is so different from "textbook" Lyme disease?

Plausible reasons for interindividual variability include bacterial inoculum size, bacterial strain differences, differences in host factors such as immune responses or genetic factors, and . . . [Full Text of this Article]

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Concurrent Infection of the Central Nervous System by Borrelia burgdorferi and Bartonella henselae: Evidence for a Novel Tick-borne Disease Complex
Eugene Eskow, Raja-Vemkitesh S. Rao, and Eli Mordechai
Arch Neurol. 2001;58(9):1357-1363.
ABSTRACT | FULL TEXT