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The Potential for Harm or Benefit From Inflammatory Processes in Stroke
John N. Whitaker, MD
Birmingham, Ala
Arch Neurol. 2001;58:674.
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| Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings. |
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GIVEN the prominent role of stroke as a cause of morbidity and mortality
throughout the world, numerous attempts have been made to prevent, limit,
and reverse its effects. In ischemic stroke, abundant effort has been placed
on recognizing risk factors and the use of medications or surgical methods
to retain or reestablish vascular patency, retard tissue damage, and promote
recovery. In these two position papers, the involvement of inflammatory mediators
in stroke is addressed. Both emphasize the need to focus on the secondary
role of inflammatory processes in a disorder usually initiated by noninflammatory
mechanisms.
A similar consideration has become prominent in other neurological disorders,
such as Alzheimer disease.1, 2
In spite of shared attention to inflammation, very divergent positions are
proposed on how to view and correctly alter the cascade of inflammatory processes
unleashed by ischemic infarction of the brain. del Zoppo et al take a . . . [Full Text of this Article]
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