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Marc I. Chimowitz, MB, ChB
From the Department of Neurology, Emory University, Atlanta, Ga.

Corresponding author: Marc I. Chimowitz, MB, ChB, Department of Neurology, Emory Clinic, fourth Floor, Clinic A Bldg, 1365 Clifton Rd, Atlanta, GA 30322 (e-mail: mchimow@emory.edu).

Arch Neurol. 2001;58:1690-1692.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

ATHEROSCLEROTIC stenosis of the major intracranial arteries is an important cause of stroke, accounting for approximately 5% to 10% of ischemic strokes in the United States.^{1, 2, 3, 4} Treatment of patients with intracranial arterial stenosis has traditionally consisted of antithrombotic therapy (antiplatelet agents or anticoagulation) and management of vascular risk factors.⁵ More recently, angioplasty or stenting have emerged as potential therapeutic options. Early experience during the 1980s showed that intracranial angioplasty was associated with an unacceptably high risk of stroke or death, and the procedure was abandoned.⁶ However, the success of coronary angioplasty and stenting, coupled with advances in microcatheter and balloon technology throughout the past decade, has led to renewed interest in intracranial angioplasty and stenting.^{7, 8, 9, 10, 11, 12, 13, 14, 15, 16, 17, 18, 19}

In assessing whether it is appropriate to use angioplasty or stenting as a first-line therapy for intracranial arterial stenosis, several questions need to be answered. (1) What is the risk . . . [Full Text of this Article]

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