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Neurological Manifestations in Sjögren Syndrome
Catherine Lafitte, MD, PhD
Arch Neurol. 2000;57:411-413.
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INTRODUCTION
Sjögren syndrome (SS) is a systemic autoimmune inflammatory disorder characterized by lymphocytic infiltration of exocrine glands with absent or diminished glandular secretion. The salivary and lacrimal glands are primarily involved, leading to dry eyes (xerophthalmia with keratoconjunctivitis sicca) and mouth (xerostomia). Cases of xerophthalmia and/or xerostomia were first mentioned in the late 19th and early 20th centuries. In 1933, studying these symptoms and their related manifestations, Sjögren,1 the Swedish ophthalmologist, concluded that they were manifestations of a general disease. Since then, the syndrome including xerophthalmia with keratoconjunctivitis and xerostomia bears Sjögren's name.
Because systemic complications of SS were not thoroughly studied before 1960, these seminal citations on neurological manifestations in SS refer to relatively recent work. Some important aspects of neurological complications in SS were accurately addressed only in 1986.
NEUROLOGICAL MANIFESTATIONS
The first author to mention neurological manifestations in SS was Sjögren himself2 in 1935. Data were . . . [Full Text of this Article]
LABORATORY FINDINGS
PATHOLOGICAL FINDINGS
PATHOGENESIS
FURTHER PERSPECTIVES
From the Service de Neurologie, Hôpital de la Salpêtrière, Paris, France.
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