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A Neurotherapeutic Advance

Henry J. M. Barnett, MD; Heather E. Meldrum

Arch Neurol. 2000;57:40-45.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

INTRODUCTION

Egaz Moniz,¹ the pioneer of angiography, in the 1920s identified carotid artery lesions in patients who had experienced a stroke. In the early 1940s, Hultquist² published early pathological observations. It remained for C. Miller Fisher's³ meticulous observations in the 1950s to focus attention on the importance of the extracranial portion of the carotid arteries as a prominent cause of ischemic stroke. Quickly the concept evolved of transient ischemic attacks as forerunners of ischemic infarction. Because surgeons had learned to repair arteries in the battlefields of World War II, it was predictable that pioneers would attempt to remove offending carotid arterial lesions. First to publish was the English team of Eastcott et al.⁴ An Argentinean and Americans missed the accolades of primacy by delaying publication.^5-6

Fields and colleagues⁷ had the vision in the early days of randomized trials to assign patients with cerebral . . . [Full Text of this Article]

THE EVIDENCE FROM THE TRIALS ON SYMPTOMATIC PATIENTS

THE EVIDENCE FROM THE TRIALS ON ASYMPTOMATIC INDIVIDUALS

THE IMPACT OF AN OCCLUDED ARTERY IN ASYMPTOMATIC PERSONS

THE IMPACT OF STROKE CAUSE ON RECOMMENDATIONS FOR CAROTID ENDARTERECTOMY

UNSETTLED PROBLEMS REGARDING CAROTID ENDARTERECTOMY

SUMMARY OF RECOMMENDATIONS REGARDING CAROTID ENDARTERECTOMY

From The John P. Robarts Research Institute, London, Ontario.

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